Peri-Implantitis Treatment with Autogenous Bone

17. Treatment of Peri-implantitis Using Open Flap Debridement and Iodine Solution with Autogenous Bone Graft: A Case Report By Miki Taketomi Saito, Mauro Pedrine Santamaria, Karina Gonzales Silvério, Enilson Antônio Sallum

Peri-implantitis is characterized by bone destruction around dental implants due to the host immune-inflammatory response induced by biofilm accumulation. Several approaches have been proposed to treat peri-implantitis, including mechanic debridement, antimicrobial therapy, and resective or regenerative surgical therapy. The present case report describes a peri-implantitis case treated by a surgical open flap debridement, decontamination of the implant surface with povidone-iodine and fill of the adjacent osseous defect with autogenous bone graft. After 20-month follow-up, the pocket depth reduction and radiographic fill of the defect could be observed. Therefore, it can be concluded that this therapeutic approach could promote clinical and radiographic improvements to the patient. However, more randomized controlled clinical trials are necessary for further understanding about the best approaches for the treatment of peri-implantitis.

25. En Masse Intrusion Of Maxillary Anterior Teeth Using Titanium Mini Implants As An Intraoral Anchorage: A Clinical Study By Rita Kashyap, Pooja Chavan, Ruchi Saxena, Vijay Naik

To evaluate efficacy of titanium mini implants as an effective means of anchorage for en masse intrusion of maxillary anterior teeth and to determine amount of intrusion achieved.

37. Stem Cells from Oral Tissues: State of the Art By Kelvin I. Afrashtehfar, Raul Rosales-Ibanez, Lanka Mahesh

This brief review is an update about adult stem cells obtained from oral tissues and their therapeutic use through tissue engineering techniques in order to produce the tissues needed to repair/regenerate, from bone or tooth tissue lost to an entire bioengineered tooth.

45. Serum Interleukin-12 and Interfereon-Gamma Levels in Humans with Periodontal Disease and Preeclampsia: A Pilot Study By Dan Holtzclaw

Preeclampsia is an obstetric complication that affects 7-10% of pregnant patients resulting in significant morbidity and/or mortality to the mother and fetus. Though the biologic basis for the development of preeclampsia is poorly understood, current research indicates that the disorder is a syndrome of excessive inflammatory immune responses. Recent studies have shown an association between periodontal disease and a number of systemic diseases including preeclampsia. The link between these maladies this thought to arise from immunologic responses to the chronic burden of endotoxin elicited inflammatory cytokines of periodontal disease. Previous studies examining the relationship between periodontal disease and obstetric complications such as preterm labor and preeclampsia have focused on inflammatory cytokines such as prostaglandin PGE2, interleukin-1β (IL-1β), and C-reactive protein. With current theories on the etiology of preeclampsia focusing on Th-1 cytokines such as interfereon gamma (IFN-ϒ) and interleukin-12 (IL- 12), the goal of this pilot study was to compare serum levels of IFN-ϒ and IL-12 in preeclamptic women with and without periodontal disease.