The Journal of Implant & Advanced Clinical Dentistry
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GBR with Induced Periodontal Regeneration

  • Journal:GBR with Induced Periodontal Regeneration Volume: Vol. 2, No. 1 Date: February 2010
    Authors:Ben Watkins, DDS, Euwe Egon, DDS, Giulio Rasperini, DDS, Maurizio Silvestri, DDS, Pradeep Padatrow, DDS, MSD, MPH, George Hilal, DMD, MDS, David Cagna, DDS, MS, Paul Bland, DDS

    19. Case of the Month: Site Development for Dental Implant Placement with Forced Eruption and Site Preservation

    Background: It is known that the extraction of a tooth leads to the resorption of approximately 1mm of facial bone alveolar bone. This expected resorption of bone can lead to gingival asymmetries with results that may be unacceptable to patients, particularly when the area is associated with a maxillary central incisor. In the case presented, a 22 year old female presented with a history of trauma to the right maxillary central incisor (tooth #8) due to sports related injuries, once in 2000 and the second in 2004. These injuries resulted in non- restorable external resorption of the tooth.

    25. Combined Regenerative Techniques: Guided Bone Regeneration Associated with Induced Periodontal Regeneration – Preliminary Reports

    Background: Guided Bone Regeneration (GBR) technique by means of autogenous bone graft and nonresorbable ePTFE membrane in atrophic alveolar ridge allows regeneration of bone in both vertical and horizontal dimension. The regeneration of periodontium with Induced Periodontal Regeneration (IPR) through the application of Enamel Matrix Derivative Proteins (EMD) onto the root surface, with formation of new cementum, new periodontal ligament and new bone is well known. Regeneration of bone directly against the root surface of natural teeth next to the implant site has been avoided so far.

    41. Iatrogenic Peri-Implantitis: Treatment and One to Two Year Follow up

    Background: Peri-implantitis may be associated with a number of variables ranging from patient related to iatrogenic factors. Peri-implant mucosal tissues may be more vulnerable to plaque induced inflammatory changes due to differences in gingival attachment between implants and natural teeth. Local factors that encourage bacterial growth and reduce implant integration should be considered when diagnosing and treating inflammatory lesions identified around restored implants.

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